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    Intracellular adhesion molecule-1 (ICAM-1), a cellular adhesion molecule that mediates the interaction of activated endothelial cells with leukocytes, is involved in various inflammatory and cardiovascular disorders.The relation between these markers and genetic polymorphism, however, remains to be elucidated.Some of these rats were treated with dexamethasone, a potent anti-inflammatory glucocorticoid, to inhibit inflammation and edema.Results: We demonstrate that inhibition of inflammation by treatment with dexamethasone significantly attenuated VEGF-induced pathological angiogenesis.A DRM who violates the ICAM Terms of Agreement will be denied immediately.This website adheres and patterns its operations after the Indiana State Office of Technology Policy and Procedures Guide.

    Separate analyses of neonates, children and adults demonstrate significant age-dependent discrepancies in both basal and septic levels of circulating soluble adhesion molecules.Moreover, significant independent associations were found between s ICAM-1 and smoking, insulin resistance index (HOMA IR), interleukin-6 level, and alkaline phosphatase and aspartate aminotransferase activities.In conclusion, this study revealed a significant association between the G/R241 ICAM-1 polymorphism and serum s ICAM-1 levels, probably due to the impairment in binding of ICAM-1 to leukocyte integrin Mac-1 protein.Received: December 16, 2005Accepted: January 26, 2007Published online: March 30, 2007 Issue release date: June 2007 Number of Print Pages: 9 Number of Figures: 7 Number of Tables: 0 ISSN: 1018-1172 (Print) e ISSN: 1423-0135 (Online) For additional information: Background: Infusion of exogenous vascular endothelial growth factor (VEGF) into adult brain at doses above 60 ng/day induces dramatic angiogenesis accompanied by vascular leak and inflammation.Blood vessels formed by this treatment are dilated and tortuous, exhibiting a pathological morphology.

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